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Save our Elephants

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The killing and dismembering of the tusker in Block 3 of Yala National Park recently brought tears to the eyes of elephant lovers. This poor animal stayed within the boundaries, marked by us to say “you are safe here”, and was still killed with a single shot to the head by inhuman poachers operating within the National Park. Further his small tusks were removed using a chainsaw. The tail was also removed because its hair is in high demand.

Why is this happening? Why can we not catch these culprits? We should do our best to stop these inhuman activities. Sri Lanka Army’s elite units can help the Wildlife Department in this effort.

As a local saying goes, you never get fed with watching ‘Bali and Ali’. ‘Bali’ is a traditional dance form and ‘Ali’ are elephants. An elephant herd is such a delightful sight; baby elephants always playing under the watchful eyes of mother elephants; the leader, although a bit aloof, always protecting weaker ones.

My wife Yamuna’s late maternal grandmother carried the longest registered name in Sri Lanka in her birth certificate – Akkada Mukkada Weera Rabukkada Rubuke Jayagath Diyathilaka Koralege Illanganthilaka Mudiyanselage Leelawathi Illanganthilaka. She was from a noble house of the Kandyan Kingdom, which was tasked with protecting the Buddha’s sacred Tooth Relic. She lived in a Walawwa behind the Hanguranketha Raja Maha Vihara, where the Tooth Relic was taken when Kandy fell to the British in 1815. Her relatives used to worship the Relic and offer Jasmine flowers.

You must be wondering what all these names that prefix her surname means. They denote honours conferred on her house, for bravery, by various Sinhala kings for fighting the enemy, to protect the Buddha’s Tooth Relic, which was the most important possession of any Sinhala king. These prefixes were conferred by the Head of State for long dedicated service and bravery. For example, I am Admiral Ravindra C. Wijegunaratne, WV, RWP and Bar, RSP, VSV, USP, NI (Military). These letters that affix my name denote Weerodara Vibhushanaya, Rana Wickrama Paddakkama, (twice), Rana Soora Padakkama, Vishista Seva Vibhushanaya, Uttama Sewa Paddakkama and last Nishan-e-imthiyas (Military) awarded by Pakistani Head of State.

Hanguranketha Walawwa produced beautiful ladies for generations. I used to say they were like Jasmine flowers which signify fragrance, delicacy and elegance. It is probably the merits of worshipping the Tooth Relic with jasmine flowers and offering jasmine flowers to the Temple of the Tooth. My significant other was ‘Miss Sri Lanka for Miss Asia Pacific contest 1989’. Her mother late Talatha Gunasekara (née Abeykoon) was a famous actress in the 1970s, who resembled famous Hindi actress Vyjayanthimala. My wife has photos of her playing tennis at Hillwood College, Kandy wearing a half-saree (lama saree) in 1952. She was very beautiful.

This noble family were elephant lovers for generations. When my wife’s grandmother used to visit our home with my mother-in-law. She used to tell me: “You know Ravi, I was married off to Abeykoon (grandfather) with enough land, three servants and one and a half elephants!”

My son, who was about six years old at that time would ask, “Muttattama (great-grandmother), how do you measure half an elephant? By cutting a big elephant in two? Aiyo!” And she would reply, “No my dear great-grandson, what I meant by one and a half elephants is an elephant and a baby elephant!”

In the 1940s, her younger brother who attended Kingswood College at the time suddenly disappeared. He was not to be found. They later came to know that he had gone with those who catch and tame wild elephants in the Northern and Eastern Provinces. He came back home after 30 years. We used to call him Lanka Attha (Lanka grandfather) because he knew all the jungle areas of Sri Lanka, having worked so well with wild elephant trainers, and all the tactics of taming an elephant. He loved elephants.

We, as a nation, never killed elephants. We protected them. Some were tamed and used for heavy work. Other tamed elephants and tuskers were used in processions. The tusker carrying the Buddha’s Tooth Relic during the Kandy Perahera is highly respected by the public.

We are a nation that trained elephants for war during ancient times, trained and guided by our trainers for combat. Elephantry is a term for a specific military unit that had elephant-mounted troops.

Pliny the Elder, a famous Roman author stated that in Mediterranean markets, there was high demand for Sri Lankan elephants. For example, Sri Lankan elephants were larger, fiercer and more suited for war than any other type. This superiority, as well as the proximity of supply to seaports, made Sri Lanka’s elephants a lucrative trading commodity.

When Arahat Mahinda Thera arrived in Sri Lanka to disseminate Buddhism, under the aegis of the great Indian Emperor Asoka, our King Devanampiya Tissa, (247 BC – 207 BC) invited Arahat Mahinda Thera to teach the Dhamma to his wives and court. According to the Dipawansa, the oldest record of Sri Lanka, due to the lack of space in the King’s palace to accommodate such a larger gathering, the elephant stable was cleaned and prepared for Arahat Mahinda’s sermon. This shows that even King Devanampiya Tissa kept tamed elephants and an elephant stable.

The famous battle between King Dutugemunu (101 – 77 BC) and Chola King Elara, the final battle at Vijithapura, where King Dutugemunu’s elephant Kandula killed King Elara’s elephant Maha Pabbatha, is vividly recorded in Mahawansa, a historical chronology of ancient Sri Lanka written by Buddhist monks.

My article in The Island newspaper of 29 August 29, 2020, ‘Protecting Mogul Emperor Aurangzeb’s silver coin ship’, relates how even in 1703, Mughal Commander at Coromandel, Daud Khan Panni, spent 10,500 silver coins to purchase 30 to 50 war elephants from Sri Lanka. This purchase was approved by our king at the time, Wimaladharmasuriya II, in Kandy, according to the book, ‘Mughal Warfare: Indian Frontiers to High roads to Empire 1500 to 1700’ by Jos JL Gommans, Leiden University in the Netherlands (page 122). The great Mughal Emperor Akbar (1556- 1605 AD) had 32,000 elephants in his stables, more than one third imported from Sri Lanka! Moghul Emperors were elephant lovers.

It was the British, who invaded our country, introduced elephant killing in the 1820s.

Due to the COVID-19 pandemic, my movements have been restricted to Nuwara-Eliya for months. I have no complaints, cool weather and fresh air being enjoyed by myself and my family, especially my son, who drives up to Nuwara-Eliya Golf Club, which is deserted, and plays nine holes every day, with no crowd and at his own pace.

A small sign next to the Golf Club drew my attention a few days ago. It gives directions to the grave of late Major Thomas William Rogers, who killed more than 1,400 elephants within eleven years, until he died struck by lightning at Haputale Rest House on June 7, 1845. Thousand four hundred elephants killed by one person in eleven years. What a crime!

Sir Samuel Baker of the early British colonial era, who was famous for Nile expeditions in Africa, was a notable elephant hunter in Sri Lanka, then Ceylon. One day he killed 11 elephants before breakfast and 104 elephants in three days. Colonial Office papers indicate that rewards were offered for killing 5,500 elephants during this time. Further Colonial records show that Baker complained when the Colonial Secretary reduced his rewards for killing elephants.

It is recorded that Major Skinner and Captain Gallewy killed 700 elephants each, but they were no match to Major Rogers. There is a connection between elephant killing and golf! Those days if you had a golf bag made out of the skin of a bull elephant’s penis, you were considered an important man. Your importance was further elevated if you were the hunter who killed that bull elephant. Such cruelty! Such vanity!

According to Wikipedia, King Edward VII owned a golf bag made from the skin of an elephant’s penis, a gift from an Indian Maharaja who had heard of the king’s fondness for golf and big game hunting. The elder son of Queen Victoria, who waited 60 years as Prince of Wales to become king, King Edward VII laid a golf course in Windsor and was a keen golfer.

Our elephants underwent untold suffering under the British. Major Rogers’ crimes did not sit well with Sinhala Buddhists and Tamil Hindu estate workers who worshipped Lord Ganesh, the elephant-headed Hindu God. They cursed him and he was struck dead by lightning. The story goes that even now lightning strikes his grave from time to time.

I asked how true these stories were from an old worker at the Nuwara-Eliya Golf Club. He confirmed it! Once lighting had struck the grave and even the Golf Club workshop had caught fire, he said. The poachers who killed and dismembered an innocent elephant in Yala Block 3, close to the Lord Skanda’s and Lord Ganesh’s temples, in Kataragama deserve similar punishment from God.

Please raise your voice to protect our elephants.

I will be failing in my duty if I do not acknowledge the research support provided by my son Ravi junior, a keen golfer and animal lover, for this article.



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Sat Mag

Contagion in Ceylon: Two case studies

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By Uditha Devapriya

The medical profession in Ceylon was, by the late 19th century, following patterns set down in most of the European colonies in Asia. The Civil Medical Department was set up in 1858, the Ceylon Medical College in 1870, and the Ceylon Medical Association in 1887. As with law, the practice of medicine soon came to be monopolised by the upper middle class, predominantly Burgher and Tamil.

In her extensive study of the history of the medical profession in Ceylon, Kamalika Pieris conjectures that this may have been due to the fact that unlike the Sinhala bourgeoisie, neither the Burgher nor the Tamil middle class had extensive holdings in land. In an autobiographical piece, on the other hand, Ralph Pieris observed that in colonial society, the quickest way for the middle class to guarantee their position was by ensuring their children took to professions where the duration between education and employment was brief; to give just one example, Lucian de Zilwa, divided between law and medicine, chose the latter because “I could begin to earn something the moment I received my sheepskin.”

The entrenchment of a medical elite did not contribute to a rise in health or sanitary standards in the country. The first hospitals had been built for military garrisons in Colombo, Galle, Jaffna, and Trincomalee, while the expansion of the plantation economy necessitated the establishment of hospitals to service the needs of estate labour. In both cases the aim would be to serve imperial interests. Against such a backdrop, expansions of medical institutions, quarantine centres, and isolation camps were driven by bouts of epidemics which became part and parcel of the region in the 19th century. The aim of this essay is to compare and contrast responses by officials to two such epidemics: smallpox and cholera.

Historically, the Sinhalese are said to have feared smallpox the most. Robert Knox wrote that none of the charms “successful to them in other distempers” could cure the illness, while Ribeiro described it as “the most dreaded disease among the natives.”

There’s reason to believe that while smallpox was introduced by European adventurers, it did exist in pre-colonial societies; Asiff Hussein writes of Hugh Neville describing “a gigantic Telambu tree amidst a sacred grove on the site of the Ruwanveli dagaba” which may have figured in an outbreak of a disease at the time of Arahat Mahinda. The first real account of the disease, according to Thein (1988), comes to us from a fourth century Chinese alchemist called Ko Hung; it may have existed in a mild form in fifth century India.

John Holwell was the first British official to write on the illness in the subcontinent, in 1767 in Bengal. From 1779 to 1796, there was a severe smallpox outbreak in that region, while in 1802 we read of a Swiss doctor, Jean de Carro, sending samples of a vaccine to Bombay via Baghdad. The date is significant, for it was in 1802 also that, as Ananda Meegama has noted, vaccination against smallpox commenced in Sri Lanka.

However, owing to inconsistencies in the vaccination programme, not least of which the slipshod way medical officials despatched vaccines to poorer, far flung regions, there were severe periodic outbreaks. In 1886, vaccination was made compulsory for the first time, but even then problems in the Medical Department, set up 28 years earlier, led to the disease spreading throughout the country. To prevent it would have meant enforcing an anti-venereal programme, which transpired 11 years later with the passing of the Quarantine and Prevention of Diseases Ordinance No 3 of 1897. By the 1930s, smallpox had ceased to become a serious threat; by 1993, it had been fully eradicated.

No less feared than smallpox was cholera, which like smallpox goes back many centuries in the country and the subcontinent. The Tamil goddess, Mariamman, is considered symbolic of both smallpox and cholera, while a reference in the Mahavamsa to a yakinni who in the reign of Sirisangabo caused those who came under her curse to have red eyes, and die the moment they came into contact with a patient, may indicate an outbreak in as early as third century Sri Lanka. I have noted this in my SAT MAG column last week.

Historical sources tell us of a reference in ninth century Tibet to a particularly violent illness (the first signs of which included “violent purging and vomiting”), although scholars doubt the authenticity of these texts. In any case, not for nothing did colonial officials call it the Asiatic cholera, given its ancient origins in the lower Bengal where, it must be said, we come across our earliest sources for the cult of a cholera goddess in the subcontinent. On the other hand, the destruction of the local economy, and unnatural changes in agriculture forced on farmers by British authorities, had an impact on these outbreaks: with irrigation schemes destroyed and neglected, water scarcities could only result in pestilence.

There were six cholera pandemics between 1817 and 1917. All of them were aggravated by disruptive economic and social changes unfolding in each of the periods they transpired in. All six originated from the Bengal, inevitable given the genesis of the disease in tropical, depressed climates, and they diffused beyond the region to as far as Russia, the US, and Latin America. Leaving behind heavy fatalities, each was deadlier than the last.

The first case in Sri Lanka was reported in 1818 in Jaffna. However, as Ananda Meegama has noted, the disease never became endemic in the island. It turned into a serious issue only after British colonisers, having appropriated Kandyan lands under the Crown Lands Encroachment Ordinance and Waste Lands Ordinance of 1840, imported labour from South India. The latter led to a widespread contagion: case numbers jumped from 16,869 between 1841 and 1850 to 35,811 in the following decade, with fatality rates rising from 61% to 68%.

Until the passing of the Quarantine Ordinance in 1897 — by which time five of the six cholera epidemics had passed through and devastated the country — officials took haphazard measures to control immigration of South Indian labour. Estate workers, on their way to the plantations, took the Great North Road to the hill country, which brought them into contact with locals in Jaffna and Mannar; this led to several epidemics between 1840 and 1880, after which the number of cases and fatalities began to reduce.

Still, officials remained oblivious to the need to quarantine estate workers until much later, a problem compounded by the fact that many such workers, even after restrictions had been imposed, came disguised as traders, an issue attributable as much to the immigrants as it was to “the partial manner” in which officials enforced the quarantine.

Two years after the passage of the Ordinance, colonial administrators shut down the Great North Road to estate labour. Those who arrived in the country were taken to Ragama. Yet despite such measures, other problems cropped up: as Kamalika Pieris has pointed out, there was widespread hostility to Western medicine, particularly in economically backward regions which doctors never attempted to reach. Contaminated water became a serious issue even in Colombo, as did unplanned urbanisation, while quarantines led those fearing for their lives to flee to other districts, spreading the disease even more.

De Silva and Gomez (1994) have argued that one of the contributing factors to the recession of these diseases were the advances made among the local population in sanitation. The first soap was imported to the island in 1850; until then, only vegetable oils had been widely used. The setting up of various Committees to probe into these outbreaks would have been another factor. Despite this, though, the fact remains that while it had the wherewithal to expand into less well off regions, the government chose for crude reasons of economy not to.

By the 1930s, at which point cholera had been significantly contained — in 1946, two years before independence, only two cases cropped up — the rise in a radical left movement among professionals, including doctors and lawyers, led to a more proactive approach being taken to pandemics and contagions. The apogee of this trend, no doubt, was the malaria epidemic of the 1930s, in which the work of volunteers was done, not by the English speaking medical elite, but by a group of young leftists, among them perhaps the first Western doctor to don the national dress, S. A. Wickramasinghe. But that’s another story.

The writings of S. A. Meegama, Asiff Hussein, Kamalika and Ralph Pieris, Robert Pollitzer, Ariyaratne de Silva, Michael G. Gomez, and M. M. Thein were used for this article.

The writer can be reached at udakdev1@gmail.com

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Sat Mag

The Remedy. Remedy?

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By Dr F E Dias Introduction

A solution to the Covidian problem was eagerly awaited – and emerged in a legion of vaccines. The development of vaccines to coronaviruses has been difficult due to vaccine-induced enhanced disease responses evident in animal studies. Antibody-dependent enhancement may be involved in the clinical observation of increased severity of symptoms associated with early high levels of SARS-CoV-2 related antibodies in patients. The gene-based vaccines dispense of the need of the wild virus and instead get the inoculated persons to produce multi-trillions of its fusogenic spike protein which by itself is a pathogen that biodistributes itself and potentially harms other parts of the body away from where the injection occurred. And yet, many experimental vaccine products were hastily unleased globally without the pivotal Phase 3 trials concluded or validated.

Front Runners

Israel and the Seychelles had high proportions of their populations vaccinated before other nations caught up. Let us compare sets of data that are not usually presented synchronously, such as in Figures 1 & 2, to observe what happened and is happening in Israel and Seychelles.

EU

The 450 million strong EU consisting of 60% of Europe’s estimated 750 million inhabitants, reported administering 522.4 million doses of vaccines as of August 14th 2021, with over 75% of its citizens receiving at least one dose of an experimental CoViD vaccine.

The EnduraVigilance system is the EU-wide database for recording vaccine injury reports, as well as other medicine-induced injuries, corresponding to the US Vaccine Adverse Events Reporting System (VAERS). EnduraVigilance data indicate, since beginning of the vaccination campaign last year through to August 14th, over two million (2,074,410) reports of vaccine-related injuries, including 21,766 deaths across the 27 member

states.

Approximately half of all reports (1,021,867) were of serious injuries, classified by this EU agency as corresponding to “a medical occurrence that results in death, is life-threatening, requires inpatient hospitalisation, results in another medically important condition, or prolongation of existing hospitalisation, results in persistent or significant disability or incapacity, or is a congenital anomaly/birth defect.”  The highest rate of problematic developments among their four emergency-authorised vaccines occurred, following the use of the Oxford-AstraZeneca product, which is reportedly linked to 947,675 injuries within the EU.

Reports of thrombotic and thromboembolic post-vaccination events among the vaccinated triggered concern regarding post-vaccination donation of substances of human origin (SoHO), such of blood, plasma, organs and tissues that may contain the pathogenic spike protein of recently vaccinated donors.

UK

A groundbreaking paper by the prestigious Oxford University Clinical Research Group, published on August 10th in The Lancet found that vaccinated individuals more than 250 times the load of Covid-19 viruses in their nostrils compared to the unvaccinated. The authors suggest that while moderating the symptoms of infection, the vaccine allows vaccinated individuals to carry unusually high viral loads without becoming ill at first, potentially transforming them into pre-symptomatic super-spreaders, and they suggest that this may contribute to the post-vaccination surges in heavily vaccinated populations globally.

Public Health England (PHE), England’s public health policy department released a report on August 6th detailing the spread of the Delta variant of the virus which includes hospitalisations and deaths where Covid-19 was a factor, between February 1st and August 2nd 2021.

It shows that 65% of hospitalisations and deaths involving Covid-19 are among those who have had at least one dose of the experimental vaccines.  Though the unvaccinated category accounts for around half of overall Delta Covid-19 infections in England, the rate of death in this group is lower than among those who received vaccines.

Considering the fully vaccinated group on its own, the PHE data show 1,355 of 47,008 identified infections were admitted to hospital, which is 2.9%, suggesting that the double-jabbed face a nearly 50% greater chance of being hospitalised if they contract CoViD-19, compared with those who have not been vaccinated.  Further, those who contracted the virus within 21 days of their first shot demonstrated a 0.97% hospitalisation rate, and those who tested positive after three weeks from their first shot demonstrated a 1.14% hospitalisation rate, indicating that the likelihood of hospitalisation is greater for the double-jabbed when compared with the single-jabbed.

North America

The VAERS, jointly run by both the Food and Drug Administration and the Centers for Disease Control and Prevention is the primary US government-funded system for reporting adverse vaccine reactions in the US.  Not all adverse events are reported into it by physicians and the VAERS report is understood to be a substantial underestimate. While 328.9 million Covid vaccine doses had been administered as of July 2nd 2021, between December 14th 2020 and July 2nd 2021, a total of 438,441 adverse events were reported to VAERS, including 9,048 deaths, 22% of which occurred within 48 hours of vaccination, and 37% occurred in people who became ill within 48 hours of being vaccinated.

2,678 pregnant women reported adverse events related to CoViD vaccines, including 994 reports of miscarriage or premature birth. VAERS also reports the deaths of two breast-feeding babies in March and July 2021 due to blood clots subsequent to the mother’s reception of the vaccine. Of the 4,456 cases of Bell’s Palsy reported, 398 reports of Guillain-Barré Syndrome. There were also 121,092 reports of anaphylaxis, 8,256 of blood clotting disorders and 1,796 cases of myocarditis and pericarditis. The VAERS data showed that 22% of deaths were related to cardiac disorders.

By the end of August 2021, the total deaths reported had exceeded 13,000 and reports of harm exceeded 600,000. With over 5000 reports of myocarditis as of August 20th, Pfizer added to their product fact sheet that “post-marketing data demonstrate increased risks of myocarditis and pericarditis, particularly within seven days following the second dose. The observed risk is higher among males under 40 years of age than among females and older males. The observed risk is highest in males 12 through 17 years of age”, admitting also that potential long-term sequalae are unknown.

The Public Health Agency of Canada (PHAC) in July estimated the rate of vaccine-related blood clotting in Canadians who have received the AstraZeneca vaccine, and said there have been 27 confirmed cases to date in Canada, with five deaths among those cases.

Blood-clotting events that are reported are the larger ones that can be detected using MRI or CT scans. However, with the RNA and viral vector vaccines, there is a new phenomenon of micro blood clots, diagnosable via D-dimer tests. These microscopic blood clots could be caused by the vaccine-generated spike proteins altering the vascular epithelia, particularly affecting their interactions with platelets in capillaries.  Some parts of the body like the brain, spinal cord, heart and lungs cannot re-generate when their tissues are damaged by blood clots. Particularly in the lungs, this may cause pulmonary hypertension that may lead to heart failure years later.

In Brief:

Fatality, Natural Immunity and Treatment

The fatality rate in the age-group up to 20, among those who actually become infected is 0.003% which suggests a five times greater likelihood of death in a road traffic accident further to an infection of CoViD, and that for those 20-40 is 0.02%. A study published on August 24th 2021 by Israeli researchers looking at over 670,000 vaccinated and unvaccinated individuals conclude that “that natural immunity confers longer lasting and stronger protection against infection, symptomatic disease and hospitalisation caused by the Delta variant of SARS-CoV-2, compared to the BNT162b2 two-dose vaccine-induced immunity”. Dr Robert Malone the virologist and immunologist credited with pioneering RNA transfection has stated that natural immunity is twenty times more protective than the vaccines. India achieved success by encouraging anti-viral medication.

Sri Lanka

Sri Lanka’s vaccinations and deaths data is shown in Figures 4 and 5.

Conclusion

Correlations in Sri Lanka’s data are apparent. Even if the preceding database reports and scientific theory are disregarded, and the global experience is ignored, it is not unreasonable to suppose that the relationships between daily deaths and daily vaccinations are causal, subject to the observed time lag. It is remarkable that the spike in total number of deaths corresponds to the spike in the total number vaccinations. It is contrary to the expert wisdom that among the Covid deaths in Sri Lanka, the proportion of deaths of vaccinated people is steadily increasing as the vaccination campaign progresses.

There is evidence that the immediate and potential sequential harm due to these vaccines may exceed the risks associated with the disease.

Cytokine storm model of Castelli et al, Front. Immunol, 2020 (above)

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Sat Mag

Brief history of plagues and pandemics

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Uditha Devapriya

By the 14th century, trade routes between the East and West had made it easier for pandemics to spread, while conquests by the Spanish and the Portuguese in the 15th and 16th centuries would introduce several diseases to the New World. Trade and colonialism hence became, by the end of the Renaissance, the main causes of plague, which scientific advancement did little to combat, much less eliminate: a physician in the 17th century would have been as baffled or helpless as a physician in the 14th or 15th in the face of an outbreak.

No doubt rapid urbanisation and gentrification had a prominent say in the proliferation of such outbreaks, but among more relevant reasons would have been poor sanitary conditions, lack of communication and accessibility, and class stratifications which excluded the lower orders – the working class as well as peasants in the colonies – from a healthcare system that pandered to an elite minority. By 1805, the only hospitals built in Ceylon were those serving military garrisons in places like Colombo, Galle, and Trincomalee.

Among the more virulent epidemics, of course, was the notorious plague. Various studies have tried to chart the origins and the trajectory of the disease. There were two outbreaks in Rome: the Antonine Plague in 165 AD and the Justinian Plague in 541 AD. With a lack of proper inscriptional evidence, we must look at literary sources: the physician Galen for the Antonine, and Procopius and John of Ephesus for the Justinian.

Predating both these was an outbreak reported by the historian Thucydides in 430 BC Rome, but scholars have ascertained that this was less a plague than a smallpox contagion. In any case, by 541 AD plague had become a fact of life in the region, and not only in Pagan Rome; within the next few years, it had spread to the Arabic world, where scholars, physicians, and theologians tried to diagnose it. Commentaries from this period tell us of theologians tackling a religious crisis borne out of pestilence: in the beginning, Islamic theology had laid down a prohibition against Muslims “either entering or fleeing a plague-stricken land”, and yet by the time these epidemics ravaged their land, fleeing an epidemic was reinterpreted to mean acting in line with God’s wishes: “Whichever side you let loose your camels,” Umar I, the founder of the Umayyad Caliphate, told Abu Ubaidah, “it would be the will of God.” As with all such religious injunctions, this changed in the light of an urgent material need: the prevention of an outbreak. We see similar modifications in other religious texts as well.

Plagues and pandemics also feature in the Bible. One frequently referred to story is that of the Philistines, having taken away the Ark of the Covenant from the Israelites, being struck by a disease by God which “smote them with emerods” (1 Samuel 5:6). J. F. D. Shrewsbury noted down three clues for the identification of the illness: that it spread from an army in the field to a civilian population, that it involved the spread of emeroids in the “secret part” of the body, and that it compelled the making of “seats of skin.” The conventional wisdom for a long time had been that this was, as with 541 AD Rome, the outbreak of the plague, but Shrewsbury on the basis of the three clues ascertained that it was more plausibly a reference to an outbreak of haemorrhoids. On the other hand, the state of medicine being what it would have been in Philistine and Israel, lesions in the “secret part” (the anus) may have been construed as a sign of divine retribution in line with a pestilence: to a civilisation of prophets, even haemorrhoids and piles would have been comparable to plague sent from God.

Estimates for population loss from these pandemics are notoriously difficult to determine. On the one hand, being the only sources we have as of now, literary texts accurately record how civilians conducted their daily lives despite the pestilence, while on the other, writers of these texts resorted to occasional if not infrequent exaggeration to emphasise the magnitude of the disease. Both Procopius and John of Ephesus are agreed on the point, for instance, that the Justinian Plague was preceded by hallucinations, which then spread to fever, languor, and on the second or third day to bubonic swelling “in the groin or armpit, beside the ears or on the thighs.” However, there is another account, by Evagrius Scholasticus, whose record of the outbreak in his hometown Antioch was informed by a personal experience with a disease he contracted as a schoolboy and to which he later lost a wife, children, grandchildren, servants and, presumably, friends. It has been pointed out that this may have injected a subjective bias to his account, but at the same time, given that Procopius and John followed a model of the plague narrative laid down by Thucydides centuries before, we can consider Evagrius’s as a more original if not more accurate record, despite prejudices typical of writers of his time: for instance, his (unfounded) claim that the plague originated in Ethiopia.

Much water has flowed through the debate over where the plague originated. A study in 2010 concluded that the bacterium Yersinia pestis evolved in, or near, China. Historical evidence marshalled for this theory points at the fact that by the time of the Justinian plague the Roman government had solidified links with China over the trade of silk. Popular historians contend that the Silk Road, and the Zheng He expeditions, may have spread the contagion through the Middle East to southern Europe, a line of thinking even the French historian Fernand Braudel subscribed to in his work on the history of the Mediterranean. However, as Ole Benedictow in his response to the 2010 study points out, “references to bubonic plague in Chinese sources are both late and sparse”, a criticism made earlier, in 1977, by John Norris, who observed that it is likely that literary references to the Chinese origin of the plague were informed by ethnic and racial prejudices; a similar animus prevailed among the early Western chroniclers against what they perceived as the “moral laxity” of non-believers.

A more plausible thesis is that the bacterium had its origins around 5,000 or 6,000 years ago during the Neolithic era. A study conducted two years ago (Rascovan 2019) posits an original theory: that the genome for Yersinia pestis emerged as the first discovered and documented case of plague 4,900 years ago in Sweden, “potentially contributing” to the Neolithic decline the reasons for which “are still largely debated.” However, like the 2010 study this too has its pitfalls, among them a lack of the sort of literary sources which, however biased they may be, we have for the Chinese genesis thesis. It is clear, nevertheless, that the plague was never at home in a specific territory, and that despite the length and breadth of the Silk Road it could not have made inroads to Europe through the Mongol steppes. To contend otherwise is to not only rebel against geography, but also ignore pandemics the origins of which were limited to neither East and Central Asia nor the Middle East.

Such outbreaks, moreover, were not unheard of in the Indian subcontinent, even if we do not have enough evidence for when, where, and how they occurred. The cult of Mariammam in Tamil Nadu, for instance, points at cholera as well as smallpox epidemics in the region, given that she is venerated for both. “In India, a cholera-like diarrheal disease known as Visucika was prevalent from the time of the Susruta“, an Indian medicinal tract that has the following passage the illness to which reference is made seems to be the plague:

Kakshabhageshu je sfota ayante mansadarunah
Antardaha jwarkara diptapapakasannivas
Saptahadwa dasahadwa pakshadwa ghnonti manavam
Tamagnirohinim vidyat asadyam sannipatatas

Or in English, “Deep, hard swellings appear in the armpit, giving rise to violent fever, like a burning fire, and a burning, swelling sensation inside. It kills the patient within seven, 10, or 15 days. It is called Agnirohini. It is due to sannipata or a deranged condition of all the three humours, vata, pitta, and kapha, and is incurable.”

The symptoms no doubt point at plague, even if we can’t immediately jump to such a conclusion. The reference to a week or 15 days is indicative of modern bubonic plague, while the burning sensation and violent fever shows an illness that rapidly terminates in death. The Susruta Samhita, from which this reference is taken, was written in the ninth century AD. We do not have a similar tract in Sri Lanka from that time, but the Mahavamsa tells us that in the third century AD, during the reign of Sirisangabo, there was an outbreak of a disease the symptoms of which included the reddening of the eyes. Mahanama thera, no doubt attributing it to the wrath of divine entities, personified the pandemic in a yakinni called Rattakkhi (or Red Eye). Very possibly the illness was a cholera epidemic, or even the plague.

China, India, and Medieval Europe aside, the second major wave of pandemics came about a while after the Middle Ages and Black Death, and during the Renaissance, when conquerors from Spain and Portugal, having divided the world between the two countries, introduced and spread diseases to which they had become immune among the natives of the lands they sailed to. Debates over the extent to which Old World civilisations were destroyed and decimated by these diseases continue to rage. The first attempts to determine pre-colonial populations in the New World were made in the early part of the 20th century. The physiologist S. F. Cook published his research on the intrusions of diseases from the Old World to the Americas from 1937. In 1966, the anthropologist Henry F. Dobyns argued that most studies understated the numbers. In the 1930s when research on the topic began, conservative estimates put the North American pre-Columbine population at one million. Dobyns upped it to 10 million and, later, 18 million; most of them, he concluded, were wiped out by the epidemics.

And it didn’t stop at that. These were followed by outbreaks of diseases associated with the “white man”, including yaws and cholera. Between 1817 and 1917, for instance, no fewer than six cholera epidemics devastated the subcontinent. Medical authorities were slow to act, even in Ceylon, for the simple reason that by the time of the British conquest, filtration theory in the colonies had deemed it prudent that health, as with education, be catered to a minority. Doctors thus did not find their way to far flung places suffering the most from cholera, while epidemics were fanned even more by the influx of South Indian plantation workers after the 1830s. Not until the 1930s could authorities respond properly to the pandemic; by then, the whole of the conquered world, from Asia all the way to Africa, had turned into a beleaguered and diseased patient, not unlike Europe in the 14th century.

The writer can be reached at udakdev1@gmail.com

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